Converting enzyme inhibition causes hypocitraturia independent of acidosis or hypokalemia
نویسندگان
چکیده
منابع مشابه
Occult causes of hypokalemia.
Most causes of hypokalemia are obvious from the clinical picture and the values for plasma electrolytes. In cases with obscure etiology, a pretreatment estimation of urinary potassium and chloride often helps lead the clinician into new and fruitful avenues of clinical investigation. Here we present four cases of hypokalemia in which the pathogenesis was initially enigmatic but was clarified so...
متن کاملRole of Tissue Angiotensin-Converting Enzyme Inhibition
Background Chronic treatment with high doses of angiotensin-converting enzyme (ACE) inhibitors prolongs survival after myocardial infarction. Since the plasma reninangiotensin system (RAS) is not consistently activated in the chronic phase after myocardial infarction, the beneficial effects of ACE inhibition have been attributed, in part, to inhibition of an activated tissue RAS. However, a rel...
متن کاملEffects of Chronic Angiotensin Converting Enzyme Inhibition
Background. Previous studies have shown that global left ventricular function is depressed after myocardial infarction. However, little is known about the effects of myocardial infarction on contractility and the passive-elastic properties of residual myocardium. Methods and Results. We evaluated isometric function and passive myocardial stiffness in isolated, noninfarcted left ventricular papi...
متن کاملRespiratory effects of angiotensin converting enzyme inhibition.
Angiotensin converting enzyme (ACE) inhibitors have been available for the treatment of hypertension and heart failure for several years. This class of drugs is generally well accepted by the patient (1), but an unusual, infrequent, and troublesome side-effect, cough, has become apparent. This article reviews this side-effect and also the possibility of other respiratory effects of ACE inhibition.
متن کاملConverting enzyme inhibition and the kidney.
We review information on the renal response to converting enzyme inhibition, and attempt to evaluate the evidence that a reduction in angiotensin II formation is responsible for the renal response. There is little response to converting enzyme inhibitors in animals or man when the renin-angiotensin system is suppressed by a literal sodium intake. With restriction of sodium intake, an increase i...
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ژورنال
عنوان ژورنال: Kidney International
سال: 1998
ISSN: 0085-2538
DOI: 10.1046/j.1523-1755.1998.00150.x